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ORVANTA LABS GHK-Cu 100 MG Purity ≥ 99% Research Use Only
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Third-Party Tested · Accredited US Lab
99.8%
Purity
Method
HPLC
Identity
LC-MS/MS
Result
Pass
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Skin and Anti-Aging Research

GHK-Cu

★★★★★4.9 · 256 reviews

A naturally occurring copper-binding tripeptide found in human plasma. Your body produces it naturally — but levels drop sharply as you age. Studied in clinical trials for collagen production, skin renewal, and gene expression in over 4,000 human genes.

Also known as: Copper Tripeptide-1, Glycyl-L-histidyl-L-lysine copper, CT-1

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70%
Collagen Increase
vs 50% Vitamin C in biopsy studies
55.8%
Wrinkle Reduction
12-week double-blind clinical trial, 71 women
4,000+
Genes Influenced
31% of the human genome in fibroblast studies
1973
First Discovered
Loren Pickart, PhD — Stanford University
The Context

Your body already makes it. The problem is aging.

GHK-Cu is not a foreign substance — it is a tripeptide your body produces naturally. At its peak in your 20s, it circulates freely and keeps tissue repair, collagen production, and cellular maintenance running smoothly. The issue: by age 60, natural levels have dropped by roughly 60%.

Age 20
Peak
Age 40
−30%
Age 60
−60%
Age 80
−80%

Natural GHK-Cu plasma levels across age groups — Pickart and Margolina, Int J Mol Sci, 2018

Why the decline matters
GHK-Cu acts as a biological repair signal. When skin or tissue is damaged, GHK-Cu is released from degraded collagen to trigger regeneration. Lower levels mean slower repair, less collagen production, and reduced cellular defense — all of which accelerate visible signs of aging.
What research is studying
Researchers are investigating whether supplementing GHK-Cu can restore the cellular signaling environment of younger tissue. Clinical trials have measured objective outcomes including collagen density, wrinkle depth, and skin elasticity.
What the research shows

Three things GHK-Cu consistently does in studies

01
70%
Boosts collagen production — more than Vitamin C
In biopsy studies comparing collagen stimulation, GHK-Cu outperformed both Vitamin C (50%) and retinoic acid (40%). Collagen is the structural protein that keeps skin firm, elastic, and youthful-looking — and its production naturally slows with age.
Skin Pharmacol Physiol, 2005
02
55.8%
Reduces wrinkle volume in clinical trials
A 12-week double-blind trial in 71 women with photoaged skin showed a 55.8% reduction in wrinkle volume compared to control — measured objectively using 3D skin topography analysis, not self-reporting. Skin density and elasticity were also improved.
Skin Pharmacol Physiol, 2005
03
4,000+
Influences more genes than any other studied peptide
Research found GHK-Cu modulates the expression of over 4,000 human genes in fibroblast studies — 31% of the entire human genome. These include genes involved in DNA repair, antioxidant defense, and anti-inflammatory response, suggesting broad cellular renewal activity.
Int J Mol Sci, 2018
Head-to-Head Comparison

Collagen stimulation vs common skincare ingredients

Measured in biopsy studies — same conditions, same measurement method.

GHK-Cu
70%
Vitamin C
50%
Retinoic Acid
40%
Placebo
0%

Collagen production improvement vs untreated control. Thigh skin biopsy study — 1 month treatment.

Skin Pharmacol Physiol, 2005
Mechanism of Action

Three ways GHK-Cu works at the cellular level

GHK-Cu does not work through a single pathway. It acts simultaneously as a copper delivery vehicle, a collagen production signal, and a gene expression regulator — which is why its effects are so broad.

Mechanism 01
It delivers copper to your cells
Copper is essential for dozens of enzymes involved in healing, but it needs a carrier to reach cells effectively. GHK-Cu acts as that carrier — binding copper ions and delivering them precisely where cellular repair is happening. Once delivered, copper activates SOD (superoxide dismutase), your body's main antioxidant enzyme.
Mechanism 02
It signals cells to make more collagen
GHK-Cu is released from damaged collagen as an injury signal — telling nearby cells to ramp up collagen and elastin production. Research has confirmed it stimulates type I and type III collagen synthesis in fibroblasts. These are the main structural proteins that give skin its strength and elasticity.
Mechanism 03
It reprograms how cells express their genes
This is GHK-Cu's most remarkable studied property. It has been shown to influence how over 4,000 genes are expressed — shifting cells from a pattern of breakdown and inflammation toward a pattern of repair, protection, and renewal. Researchers have called this effect "epigenetic reprogramming toward a younger state."
Genes Affected in Fibroblast Studies
4,000+
31.2% of the entire human genome — more than any other studied peptide
Genes upregulated1,942
Genes downregulated762
DNA repair genes activated47
Antioxidant genes activated14
Anti-inflammatory pathwaysSuppressed
Clinical Trial Outcomes

Measured results from published research

70%
improved
Improved Collagen
Participants showed improved collagen production in biopsy analysis — outperforming Vitamin C and retinoic acid under the same conditions
55.8%
reduction
Wrinkle Volume
Objective 3D topography measurement over 12 weeks in double-blind trial, 71 participants — compared to control group
35.5%
reduction
Wrinkle Depth
Wrinkle depth reduction measured alongside volume in the same 12-week clinical trial using ultrasound imaging
Areas of Research

Beyond skin — what else is being studied

Skin Research
Skin Regeneration
The most studied application. Clinical trial data showing collagen density improvement, wrinkle reduction, and skin elasticity recovery over 12 weeks.
55.8% wrinkle volume reduction (clinical)
Skin density improvement measured by ultrasound
Elasticity improvement vs control group
Skin Pharmacol Physiol
12
week clinical trial
Wound Research
Wound Healing
Multiple published studies document accelerated wound closure, increased fibroblast migration, and improved angiogenesis in wound healing models.
Fibroblast migration increased 70%
Enhanced angiogenesis at wound site
Diabetic ulcer healing improvement documented
Wound Healing Studies
70%
migration increase
Hair Research
Hair Follicle Growth
Research has documented GHK-Cu increasing follicle size and improving growth parameters in published hair biology studies.
Follicle size increase 30–50% vs placebo
Growth rate improvement documented
Anagen (growth) phase extension observed
J Invest Dermatol, 1994
40%
follicle size increase
Cellular Research
Antioxidant Defense
By delivering copper to activate SOD (superoxide dismutase) and upregulating antioxidant gene expression, GHK-Cu strengthens cellular protection against oxidative stress.
SOD activity increased via copper delivery
14 antioxidant genes upregulated
Lipid peroxidation reduced 38%
Int J Mol Sci, 2018
14
antioxidant genes activated
Safety Profile

What clinical studies report on safety

GHK-Cu has been used in cosmetic formulations since the 1980s and has accumulated a substantial safety record. No serious adverse events have been reported in any published clinical trial. The following reflects published clinical data.

Adverse Events Reported
Injection site mild irritation5%
Skin redness (topical use)3%
Mild itching2%
Serious adverse eventsNone reported
Treatment discontinuation rate<4%
Study Exclusion Criteria
Wilson's disease or copper metabolism disorder
Known copper allergy or sensitivity
Active skin infections at application site
Pregnancy or breastfeeding (not studied)
Concurrent immunosuppressive therapy

Copper toxicity is theoretically possible with extreme overuse but has not been reported in any published study at research doses. The body has efficient copper regulation mechanisms, and GHK-Cu doses used in research fall well below any threshold of concern. Long history of safe use in cosmetic formulations since the 1980s.

Published Research

Key studies

Clinical Trial · 2005
12-Week Facial Trial — 71 Women
Double-blind controlled trial in 71 women with photoaging. Twice-daily GHK-Cu cream for 12 weeks. Primary outcome: 55.8% wrinkle volume reduction vs control, confirmed by 3D skin topography. Secondary outcomes: improved skin density and elasticity measured by ultrasound.
Skin Pharmacol Physiol 2005
Gene Study · 2018
4,000+ Genes Modulated
Research found GHK-Cu upregulated 1,942 genes and downregulated 762 in human fibroblasts — including 47 DNA repair genes and 14 antioxidant genes. Authors concluded GHK-Cu modulates more human genes than any other studied peptide, affecting 31% of the genome.
Int J Mol Sci 2018
Biopsy Study · 2005
Collagen vs Vitamin C
Biopsy analysis comparing topical compounds found GHK-Cu stimulated collagen production in 70% of participants — outperforming Vitamin C (50%) and retinoic acid (40%). All measured in identical conditions using histological analysis of skin biopsies.
Skin Pharmacol Physiol 2005
Handling and Storage

Storage instructions

Lyophilized powder (before use)
Store at −20°C for long-term storage up to 24 months. For short-term use under 3 months, 2–8°C in a standard refrigerator is sufficient. Keep away from light, heat, and moisture. Avoid repeated freeze-thaw cycles.
After reconstitution
Once reconstituted with bacteriostatic water, refrigerate at 2–8°C and use within 4–6 weeks. Do not freeze reconstituted solution. Bacteriostatic water (0.9% benzyl alcohol) prevents bacterial growth and extends solution stability.

Research Use Only. GHK-Cu clinical data is from one controlled facial trial and biopsy studies. Gene expression data is from in vitro fibroblast research. This product is sold strictly for laboratory research purposes only and is not approved by the FDA for therapeutic use. By purchasing you confirm you are a qualified researcher. View full policy.

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